If the full total results in animals end up being similar in humans.

The consequences persisted at two months of age. These results claim that if we prevent obesity, inflammation and oxidative stress in pregnant animals, we can prevent obesity in the offspring, stated Simmons. Simmons added that a next thing in this study is to determine the mechanisms by which inflammation and oxidative stress cause more fat cells to develop. She cautioned that until these results are studied in people carefully, one shouldn’t conclude that the biological effects seen in animals are the same in human beings. For the time being, she added that, whether pregnant or not, ladies should certainly not conclude from this scholarly research that they should consume large doses of antioxidant vitamins..The slower beating rate was because of a limited group of changes in the actions potential waveform, the electrical signal that is generated by the cells. The changes were caused by modified behavior of some ion stations in the membranes of the older cells. Like most initial discoveries in fundamental science, this study opens many more questions and avenues for additional research. But the significance of the analysis is that it increases the possibility that sinoatrial ion stations and the signaling molecules that regulate them could be novel targets for medications to slow the increased loss of aerobic capacity with age group.